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Research: HOFMANN and TSCHOP
Listed in Issue 180
Abstract
HOFMANN and TSCHOP, Department of Medicine, Obesity Research Center, University of Cincinnati College of Medicine, Ohio, USA demonstrate that fructose rather than glucose-sweetened beverages impaired insulin sensitivity and increased fat in overweight or obese adults.
Background
Coronary heart disease is a major cause of morbidity and mortality in Western societies.
Methodology
The metabolic syndrome, characterized by obesity, insulin resistance, elevated blood pressure, elevated triglycerides, and low levels of high-density lipoprotein cholesterol, confers substantial risk of coronary heart disease.
Results
Current pathogenetic models suggest that postprandial hyperlipidemia is one specific metabolic abnormality that is typically associated with increased morbidity. In this issue of the JCI, Stanhope and colleagues demonstrate that consumption of fructose-sweetened but not glucose-sweetened beverages for 10 weeks increases de novo lipid synthesis, promotes dyslipidemia, impairs insulin sensitivity, and increases visceral adiposity in overweight or obese adults (see the related article beginning on page 1322).
Conclusion
References
Hofmann SM and Tschop MH. Dietary sugars: a fat difference. Journal of Clinical Investigation, 119(5): 1089-92. May 2009. Comment on: J Clin Invest. 119(5):1322-34; PMID: 19381015]. May 2009. Source: NLM. PMC2673865 [Available on 12/31/09].
Comment
Nutrition and dietary habits may exert a significant effect upon clinical improvement following coronary heart attack. This ought to result in the greater prominence of nutritional therapists as a profession; what makes me think that more prescriptions for cholesterol lowering drugs will be more the case rather than nutritional counselling?
